简介：Laserisusedtoproduceverypowerfulbeamsoflight,sopowerfulthattheycanmakeholesinsteelinaflash.Todaylaserisfindingwiderandwideruseinmoreandmorefields.

简介：Anewtypeofelectrochemicaloscillationinducedbysurfactantwasobservedinexperiments.TheelectrochemicalsystemisaDaniellcellwithacopperrodinCuSO4aqueousandanaluminumrodinAl(NO3)3aqueousaselectrodes.ThesurfactantsareCTAB,TX-100,SLS.Theadditionoftracesurfactantsolutionbyamicro-syringemadetheoriginalmonotonouslychangingelectrochemicalsystemproduceobviousperiodicphenomena.Atthemeantime,thecopperionselectiveelectrodeandHg2SO4referenceelectrodewereusedtomonitorthecopperelectrodereactionanddetermineitsrateconstantkoffirstorderreaction.Accordingtotheexperimentalresultsofelectrodereactionkinetics,thepossiblemechanismwasfoundtobethepolarizationinducedfromthedirectionaladsorptionoftracesurfactantontheelectrodesurface.Thatistheelectrochemicaloscillations.

简介：K.J.,K.W. and K.R. Lees,we determined whether isoflurane preconditioning reduced glutamate receptor over-stimulation-induced neuronal death. Our results showed that isoflurane preconditioning reduced the neuronal death caused by over-stimulation of the major glutamate receptors [12]. Kapinya et al should be credited as the first group to show that isoflurane preconditioning induced a delayed phase of neuroprotection [13]. Our in vivo study showed that this protection was mediated by the activation of the mitogen-activated protein kinase p38 [14]. The isoflurane preconditioning-induced protection in human neuroblastoma SH-SY5Y cells involved the extracellular signal-regulated kinase-early growth response gene 1-Bcl-2 pathway activation [15]. Our in vivo study also showed that isoflurane preconditioning induced a delayed phase of protection in immature brains [16]. This protection was sustained even when animals were examined at one month after the brain ischemia [17]. In summary

简介：Fatiguecuttingisanewapproachforseparatingmaterial.Man-madefatiguecanberealizedbyapplyingarotatingbendingloadtoanotchedbar.Tobetterutilizethenewmethod,lasertreatmentisadoptedinthisstudy.Afterlaserradiationatthenotchroot,thefatiguecycleofthebardropsdramatically.Basedontheexperimentalresult,wedrawtheconclusionthatthefatigueofthebarisinfluencedbytheshapeofthehardenedarea.Ahardenedareathathasasmallaxialdimensionandarelativelylargeradialdimensionfacilitatesthefatigue.Thedesirablehardenedareacanbeobtainedbycontrollingthelasertreatmentparameters.

简介：K.J.,K.W. and K.R. Lees,J.M.

简介：volatile anesthetics can induce acute and delayed phases of preconditioning effects in the brain. Since isoflurane is a relatively safe agent and has been frequently used in patients with potential brain ischemia,7]. We showed for the first time that preconditioning with the volatile anesthetic isoflurane induced an acute phase of neuroprotection [8]. Our subsequent study showed that this isoflurane preconditioning-induced acute phase of neuroprotection was dose-dependent with an EC50 1.17% and that preconditioning the brain with isoflurane for 15-30 min was needed for the preconditioning to be maximally protective. This effect was glutamate transporter-dependent [9]. Other volatile anesthetics,we determined whether isoflurane preconditioning reduced glutamate receptor over-stimulation-induced neuronal death. Our results showed that isoflurane preconditioning reduced the neuronal death caused by over-stimulation of the major glutamate receptors [12]. Kapinya et al should be credited as the first group to show that isoflurane preconditioning induced a delayed phase of neuroprotection [13]. Our in vivo study showed that this protection was mediated by the activation of the mitogen-activated protein kinase p38 [14]. The isoflurane preconditioning-induced protection in human neuroblastoma SH-SY5Y cells involved the extracellular signal-regulated kinase-early growth response gene 1-Bcl-2 pathway activation [15]. Our in vivo study also showed that isoflurane preconditioning induced a delayed phase of protection in immature brains [16]. This protection was sustained even when animals were examined at one month after the brain ischemia [17]. In summary

简介：Basedontheself-excitedvibrationtheoryofice,thevibrationcontroltechnologyofjacketplatformisstudiedinthispaper.Themagnetorheologicalsuspensions(MR)unitischosenasthedamper,thecontrolobjectivefunctionforvibrationexcitedbyiceisdeterminedbyinstantaneousoptimalcontrol(IOC)method,andgeneticalgorithm(GA)isusedtoselecttheoptimalcontrolforce.Forthejacketplatformof40minheightanda3-floordeck,thevibrationresponsesinducedbyicehavebeencalculatedbeforeandaftercontrolconsideringthedifferentthicknessandspeedofice.Itisshownthatthecontrolmethodpresentedinthispapercanreducethevibrationresponseby30%,anditisfeasibletoadoptMRabsorberandGAinthecontrolofvibrationinducedbyice.

简介：Ithasbeenknownforsometimethatstraininjuriesoccurthroughtheexcessivelengtheningofamuscle.~(1–3)However,theprecisemechanismforstraininjuryremainselusiveandhasbeenthesubjectofrecentdebateintheJournalofSportandHealthScience,~(4–9)butitseemssensibletoconsidermechanismsatthelevelofthemusclefiber.Itwaspreviouslybelievedthatdamagewastheresultofnon-uniformstretchingofsarcomeres

简介：Exercise-inducedrhabdomyolysis（exRML）,apathophysiologicalconditionofskeletalmusclecelldamagethatmaycauseacuterenalfailureandinsomecasesdeath.IncreasedCa2+levelincellsalongwithfunctionaldegradationofcellsignalingsystemandcellmatrixhavebeensuggestedasthemajorpathologicalmechanismsassociatedwithexRML.TheonsetofexRMLmaybeexhibitedinathletesaswellasingeneralpopulation.PreviousstudieshavereportedthatpossiblecausesofexRMLwereassociatedwithexcessiveeccentriccontractionsinhightemperature,abnormalelectrolytesbalance,andnutritionaldeficienciespossiblegeneticdefects.However,theunderlyingmechanismsofexRMLhavenotbeenclearlyestablishedamonghealthprofessionalsorsportsmedicinepersonnel.Therefore,wereviewedthepossiblemechanismsandcorrelatedpreventionofexRML,whileprovidingusefulandpracticalinformationfortheathleteandgeneralexercisingpopulation.

简介：从上个世纪至今，显示器的发展已经经历了CRT(阴极射线管)、LCD(液晶显示器)、PDP(等离子体)等几个阶段，其中各有各的优缺点，比如，CRT笨重，但色彩丰富；LCD和PDP轻薄，但色彩表现力远远逊色于CRT。本人将综合以上各种显示器的优点，试图设计一种表现更为突出的显示器，我将它命名为“激光投影显示器(LPDLaserProjectionDisplay)”，希望我能为显示器的发展做出一点微薄的贡献。

简介：由红外线的激光，表面reducibility和Cu-Cr的吸附力照耀建筑群能被改进，由于相片破碎和激光texturing的相互作用。由有约束力的精力系列和钻系列分析了，铬离子的原子价状态和铜离子分别地在放射以后是+3和+1，它仍然有reducibility释放电子。在对比与在红外线附近(NIR)1064nm并且中间红外线(米尔)在15W的一样的平均产量力量的10600nm激光，在Cu-Cr建筑群的减少的金属百分比显然与纳米在深度被区分开来到微米。在化学plating以后，平均涂层厚度和NIR样品的吝啬平方的偏差是11.61m并且0.30为铜层，和2.69m并且0.08为镍层。结果是比米尔样品的那些好一些的。

简介：在这份报纸，对工业地合适的一个灵活高精确的刻度方法被建议。并列转变的复杂性被选择照相机坐标系统为统一参考书坐标系统简化。一个灵活平面刻度模式被介绍给刻度过程，它能任意地被放并且从哪个已知的特征点能被提取构造另外的未知特征点。与已知的内在的参数，激光投影机飞机方程被multi-noncollinear点适合，它通过三角测量和生气比率的射影的不变性的原则被获得。与这个方法，严格的排列和并列转变的多重时间能被避免。试验性的结果证明根的算术平均数意味着距离的平方(RMS)错误是0.0007公里。

简介：Thesupercontinuum(SC)generationattherepetitionrateof10GHzispresented.TheSCisgeneratedina4.2kmconventionaldispersion-shiftedfiber(DSF),whichisproducedwithnonlineareffectsandgroupvelocitydispersion.TheDSFispumpedbyanactivelymode-lockedEr3+-dopedfiberlaserwithpulsewidthof7.97ps.AnovelSCpulsesourcewithabandwidthupto125nmisobtained,whichcoversthewholeC,LbandsandpartofSband.Thestable,narrowpulseswithmeanpulse-widthof9.7psandtime-bandwidthproductof0.48arefilteredoutacrossthewholeSCbandwidth.Thissupercontinuumpulsesourceissuitableforfuturehigh-speedopticalcommunications.

简介：损坏结构的碎片引起的危险在意外爆炸或敌对强风事件通常是重要的。估计可能的碎片尺寸，速度和发射距离的一个可靠、有效的方法将是有用的估计并且设计反措施减轻可能的碎片危险。这篇论文为预言尺寸和石工技术墙的爆炸损坏引起的碎片的发射距离论述一个数字方法。有不同放大距离的数字模拟被执行，并且碎片的统计分发功能缩放，发射距离以放大距离被导出。

简介：

简介：Purpose:Thepurposeofthisstudywastoexamine,usingmeta-analyticmeasures,theevidenceregardingtheoptimalexerciseintensityatwhichimprovementsinspeedofcognitivefunctionaretriggered.Specifically,itwashypothesizedthatthecatecholamine,lactate,andventilatorythresholdsisthepointatwhichsignificantimprovementsinspeedofcognitivefunctionareobserved.Methods:Wecomparedmeaneffectsizesforthresholdstudiesandforthosestudieswhereexerciseintensitywasclassedasmoderate(40%e79%VO2maxorequivalent)butinwhichthethresholdswerenotmeasured.Results:Randomeffectsmeta-analysisshowedsignificant,moderate,meaneffectsizesforstudiesatthethreshold(g=0.58,Z=2.98,p<0.003)andforthoseduringmoderateintensityexercisebutinwhichthethresholdwasnotmeasured(g=0.54,Z=5.01,p<0.001).Therewasnosignificantdifferencebetweenmeaneffectsizes,whichsuggeststhatthethresholdsareunlikelytorepresentatriggerpoint.Conclusion:Moderateintensityexercise,evenbelowthethresholds,caninduceimprovedspeedofcognition,possiblyduetoacombinationofincreasedperipheralcatecholamineconcentrationsinducingvagal/nucleustractussolitariipathwayactivationandcentralincreasesduetoperceptionsofstress.

简介：

简介：Background:Plateletrichplasma(PRP)therapyiswidelyusedinenhancingtherecoveryofskeletalmusclefrominjury.However,theimpactofintramusculardeliveryofPRPonhematologicandbiochemicalresponseshasnotbeenfullyelucidatedinexercise-inducedmuscledamage.ThepurposeofthisinvestigationtheeffectsofintramusculardeliveryofPRPonhematologicandbiochemicalresponsesandrecoverystrategymuscledamageinducedbyhighintensitymuscleexercise(exercise-inducedmuscledamage,EIMD).Methods:Moderatelyactivemalevolunteersparticipatedinthisstudyandwereassignedtoacontrolgroup(control,n=6)andPRPadministrationgroup(PRP,n=6).Thesubjectsperformedexercisewithaloadof80%onerepetitionmaximum(1RM)maximalvoluntarycontractionoftheelbowflexorsuntilpointofexhaustionofthenon-dominantarmwasreached.ThearmsweretreatedwithsalineorautologousPRPpost-24hEIMD.Venousbloodsampleswereobtainedinthemorningtoestablishabaselinevalueand1–4dayspost-exerciseandwereanalyzedforserumferritin,iron,ironbindingcapacity(IBC),creatininekinase(CK),lactatedehydrogenase(LDH),aspartateaminotransferase(AST),andalanineaminotransferase(ALT).Results:Thebaselinelevelsofplasmairon,ferritin,IBC,CK,LDH,AST,andALTweresimilarinboththecontrolandPRPgroups.However,24-hfollowingexerciseasignificantincreaseintheseparameterswasobservedinbothgroupsbetween1and4daysduringtherecoveryperiod.Interestingly,PRPadministrationdecreasedplasmaironlevelscomparedtothecontrolontheseconddaypost-exercise.PlasmaIBCincreasedinPRPgroupfromDays2to4post-exercisecomparedtothecontrolgroupwhilstPRPadministrationhadnoeffectonplasmaferritin,CK,AST,ALT,orLDH.Conclusion:Acuteexhaustiveexerciseincreasedmuscledamagemarkers,includingplasmairon,IBC,andferritinlevels,indicatingmuscledamageinducedbyexercise.PRPadministrationimprovesinflammationbyreversingtheincreaseintheironlevelspost-exercisewi