简介:关于到节肢动物害虫的主人植物抵抗的应用研究被雷金纳德画家原来在他的1951书开发的一个框架在过去的60年指导了,在庄稼植物的昆虫抵抗。画家把抵抗的现象划分了成三机制,nonpreference(以后重命名的antixenosis),抗生现象,和忍耐。这个框架的软弱被讨论。特别地,这个trichotomous框架不包含抵抗的所有已知的机制,并且antixenosis和抗生现象范畴在实践模糊、无法分开。这些特征也许导致了一条过分简单化的途径到在庄稼植物理解节肢动物抵抗。一个两分的计划作为代替被建议,与在抵抗(限制损害到植物的植物特点)和忍耐(每统一损害减少收益损失的数量的植物特点)之间的一个主要部门,并且抵抗范畴细分进组成/可诱导、直接/间接的子范畴。两分的计划仔细到更多的采用这的最重要的好处在植物抵抗上排列基本、适用的文学并且在庄稼植物种抵抗鼓励一条更机械学的途径到学习。一条更机械学的途径将被需要为把植物抵抗集成到害虫管理节目开发新奇途径。
简介:CellResearchispleasedtoannouncethatDrDangshengLihasbeenappointedasthenewDeputyEditor-in-chiefforourjournal,startingMarch2006.DangshengreceivedhisBSdegree(inCellBiology)fromtheUniversityofScienceandTechnologyofChina,andobtainedhisPhD(inMolecularBiology)fromCornellUniversityGraduateSchoolofMedicalSciences.AfterextensivepostdoctoraltrainingatNewYorkUniversityMedicalCenter,hejoinedCellPressin2004toserveasanAssociateEditorforthepremiumbiologyjournalCell.DangshengwillbeinchargeofthescientificeditingoperationsofCellRe-
简介:ItisoutofquestionthatChinahasachievedconsiderableadvancementsinscienceandtechnology.Theworldwidespreadof"MadeinChina"thatgetsonthewesterncountries'nervesisagoodillustration.However,althoughcomparativelybetterthansomeotherdevelopingcountries,wehavetofacethesituationthatscienceandtechnologyinsuchagreatnationasChinaisstillseriouslylaggedbehind,lackinggreatbreakthroughsandtop-rankingmastersandleaders.That'swhywefeeluneasyinfrontofinternationalcolleagues.
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简介:Poly(vinylalcohol)/hydroxylapatite(PVA/HA)compositehydrogelwaspreparedbyrepeatedfreezingandthawing.Thewaterlosspropertiesoftheresultanthydrogelwereinvestigatedbyusingopticalmicroscope.LongtimeimmersiontestsofPVA/HAcompositehydrogelwerecarriedoutinthedilutedcalfserumsolutiontostudythechangelawsofswellingpropertieswiththefreezing-thawingcyclesandHAcontent.Themicro-morphologiesofPVA/HAcompositehydrogelafterlongtimeimmersionwereobservedbymeansofthehigh-accuracy3Dprofiler.TheresultsshowthattheswellingprocessofPVA/HAcompositehydrogelistheconverseprocessofitswaterloss.LongtimeswellingratiocurvesofPVA/HAcompositehydrogelinthecalfserumsolutionaremanifestedasfourstagesofquickincrease,decrease,slowdecreaseandstablebalance,anditsequilibriumswellingratiodecreaseswiththeincreaseoffreezing-thawingcyclesandHAcontent.ItisrevealedthatthenetworkstructureofthecompositehydrogelimmersedforalongperiodissignificantlyimprovedwiththeincreaseofHAcontent.PerfectnetworkstructuresofPVA/HAcompositehydrogelaswellasfullandequilibriumtissuesafterswellingequilibriumareobtainedwhentheHAcontentis3%andthenumberoffreezing-thawingcyclesis7.
简介:Theheadmounteddisplay(HMD)iswidelyusedinvirtualrealitytechnology.IncommonHMD,however,thebinoculardisparityissettoanequalfixedvalueintheentirerangeofview.SuchHMDsystemshaveseveralshortcomingswhenusedforwideviews.Inthisstudy,inordertorealizeanaturalstereosensationofHMDwithwideview,wemeasurethecharacteristicsofbinocularstereoperceptionandbinocularlightperception.Resultsshowthatboththestereoacuityandlightsensitivitydecreaseastheretina'seccentricityincreasesfromfoveatoperiphery.However,thedecreaseofthestereoacuityismorerapidthanthatofthelightsensitivity.Theseresultssuggestthatthebinoculardisparityattheperipheralfieldshouldbesmall,otherwisedoubleimageswouldbeobservedinsteadofastereoview.Basedontheresultswedeveloparelativebinocularstereoacuitymodelwhichcanbeapplied{orthedesignofHMDsystemswithwideview.
简介:Humanμ-opioidreceptor(HμOR)withatagofsixconsecutivehistidinesatitscarboxylterminushasbeenexpressedinrecombinantbaculovirusinfectedSf9insectcells.Themaximalbindingcapacityforthe[^3H]diprenorphineand[^3H]ohmefentanyl(Ohm)were9.1±0.7and6.52±0.23nmol/gprotein,respectively.The[^3H]diprenorphineor[^3H]OhmbindingtothereceptorexpressedinSf9cellswasstronglyinhibitedbyμ-selectiveagonists[D-Ala^2,N-methyl-Phe^4,glyol^5]enkephalin(DAGO),Ohm,andmorphine,butneitherbyδnorbyκselectiveagonist.Na^+(100mM)andGTP(50μM)couldreduceHμORagonistsetorphineandOhmaffinitybindingtotheoverexpressedHμOR.μ-selectiveagonistsDAGOandOhmeffectivelystimulated[^35S]GTPγSbinding(EC50=2.7nMand6.9nM)andinhibitedforskolin-stimulatedcAMPaccumulation(IC50=0.9nMand0.3nM).Theagonist-dependenteffectscouldbeblockedbyopioidantagonistnaloxoneorbypretreatmentofcellswithpertussistoxin(PTX).TheseresultsdemonstratedthatHμORoverexpressedinSf9insectcellsfunctionallycoupledtoendogenousCi/oproteins.